Cerebrospinal fluid monoamine metabolites in boys with attention-deficit hyperactivity disorder
References (45)
- et al.
Aggression in humans correlates with cerebrospinal fluid amine metabolites
Psychiatry Research
(1979) - et al.
Methylphenidate and dextroamphetamine treatments of hyperactivity: Are there true nonresponders?
Psychiatry Research
(1991) - et al.
MMPI measures of impulsivity and depression correlate with CSF 5-HIAA and HVA in depression but not schizophrenia
Journal of Affective Disorders
(1991) - et al.
A longitudinal assessment of CSF monoamine metabolite and plasma cortisol concentrations in young rhesus monkeys
Biological Psychiatry
(1992) Cruelty to animals and CSF 5HIAA
Psychiatry Research
(1989)- et al.
Objective and subjective side effects of research lumbar punctures in children and adolescents
Psychiatry Research
(1988) - et al.
Concentration gradient of CSF monoamine metabolites in children and adolescents
Biological Psychiatry
(1988) - et al.
CSF somatostatin in childhood psychiatric disorders: A preliminary investigation
Psychiatry Research
(1990) - et al.
Low CSF 5-HIAA concentration differentiates impulsive from non-impulsive violent behavior
Life Sciences
(1983) - et al.
Cerebrospinal fluid homovanillic acid and 5-hydroxyindoleacetic acid in adults with attention deficit disorder, residual type
Psychiatry Research
(1984)
Cerebrospinal fluid monoamine precursor and metabolite levels in children treated for leukemia: Age and sex effects and individual variability
Biological Psychiatry
Simultaneous determination of 3-methoxy-4-hydroxyphenylglycol, 5-hydroxyindoleacetic acid, and homovanillic acid in cerebrospinal fluid with high-performance liquid chromatography using electrochemical detection
Analytical Chemistry
CSF amine metabolites in children with minimal brain dysfunction: Evidence for alteration of brain dopamine—A preliminary report
Journal of Pediatrics
Relationship between dominance hierarchy, cerebrospinal fluid levels of amine transmitter metabolites (5-hydroxyindole acetic acid and homovanillic acid) and plasma cortisol in monkeys
Neuroscience
Manual for the Child Behavior Checklist and Revised Child Behavior Profile
DSM-III: Diagnostic and Statistical Manual of Mental Disorders
DSM-III-R: Diagnostic and Statistical Manual of Mental Disorders
Correlation between cerebrospinal fluid amine metabolites and psychomotor activity in affective disorders
Journal of Neurochemistry
Aggression, suicide, and serotonin: Relationships to CSF amine metabolites
American Journal of Psychiatry
CSF serotonin metabolite (5-HIAA) studies in depression, impulsivity, and violence
Journal of Clinical Psychiatry
An ambulatory activity monitor with solid-state memory
ISA Transactions
Cerebrospinal fluid probenecid studies: A reinterpretation
Biological Psychiatry
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2019, Advances in Clinical ChemistryCitation Excerpt :Likely because of the same reasons above indicated for NE and 5-HT, research has not yet clearly indicated the magnitude and the direction of possible changes in HVA levels related to aggressive behavior. Almost half of the studies dealing with this issue did not find any significant correlation between the levels of HVA in the CSF, plasma or urine and aggressive behavior [118,119,126,130,133,134,139]. Other studies reported either a negative [122,131,141,145] or a positive [137,157] correlation between HVA levels and aggression.
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