Psychiatry Research
Volume 170, Issue 2 , Pages 271-272 , 30 December 2009

CD24 gene polymorphism is associated with the disease progression and susceptibility to multiple sclerosis in the Iranian population

Received 5 June 2008 ,Revised 18 November 2008 ,Accepted 5 January 2009.

References 

  1. Bai XF, Li O, Zhou Q, Zhang H, Joshi PS, Zheng X, et al. CD24 controls expansion and persistence of autoreactive T cells in the central nervous system during experimental autoimmune encephalomyelitis. The Journal of Experimental Medicine. 2004;200:447–458
  2. Duperray C, Boiron JM, Boucheix C, Cantaloube JF, Lavabre-Bertrand T, Attal M, et al. The CD24 antigen discriminates between pre-B and B cells in human bone marrow. Journal of Immunology. 1990;145:3678–3683
  3. Goris A, Maranian M, Walton A, Yeo TW, Ban M, Gray J, et al. CD24 Ala/Val polymorphism and multiple sclerosis. Journal of Neuroimmunology. 2006;175:200–202
  4. Kurtzke JF. Rating neurologic impairment in multiple sclerosis: an expanded disability status scale (EDSS). Neurology. 1983;33:1444–1452
  5. Liu Y, Zheng P. CD24: a genetic checkpoint in T cell homeostasis and autoimmune diseases. Trends in Immunology. 2007;28:315–320
  6. McDonald WI, Compston A, Edan G, Goodkin D, Hartung HP, Lublin FD, et al. Recommended diagnostic criteria for multiple sclerosis: guidelines from the International Panel on the diagnosis of multiple sclerosis. Annals of Neurology. 2001;50:121–127
  7. Otaegui D, Saenz A, Camano P, Blazquez L, Goicoechea M, Ruiz-Martinez J, et al. CD24 V/V is an allele associated with the risk of developing multiple sclerosis in the Spanish population. Multiple Sclerosis. 2006;12:511–514
  8. Zhou Q, Rammohan K, Lin S, Robinson N, Li O, Liu X, et al. CD24 is a genetic modifier for risk and progression of multiple sclerosis. Proceedings of the National Academy of Sciences of the United States of America. 2003;100:15041–15046

PII: S0165-1781(09)00014-6

doi: 10.1016/j.psychres.2009.01.002

Psychiatry Research
Volume 170, Issue 2 , Pages 271-272 , 30 December 2009